This question, though very worthwhile, doesn't necessarily need to be answered. Vaccines for EBV are under development. If we stop most EBV infections, we can stop most MS.
Doesn't help you if you've already got a persistent EBV infection though, and the host immune response to EBV may be something that you want to avoid stimulating if you've already got it in you and can't get it out.
Something like CRISPR/Cas9 editing may be the way to treat persistent EBV and other herpesvirus infections.
Maybe, though it appears that covid vaccines help clear long covid, so maybe not. Until we have a vaccine we cannot study this to see how or if it works.
Persistent EBV and long COVID are very unlikely to share any mechanisms, those are apples and oranges.
With little protein expression, training T-cells to identify viral MHC-epitopes is unlikely to ever clear something like EBV. There's no proteins, there's no virions, there's no proteases, the better target is the genome itself:
Right but when was the last time anyone you know got polio? Vaccines aren't 100% effective but if we can get the average susceptibility to the virus in the population down far enough (even though it'll never be complete) there's a threshold where the virus can have a hard enough time hopping that it really makes a difference.
